Endometrial Hyperplasia - Knowing Your Own Diagnosis

Endometrial Hyperplasia is a disorder that is believed to be one of the pre-disposing factors for the development of uterine cancer. This disorder is brought by different hormonal alterations in the body. The public views this disorder as cancer itself or a developing uterine anomaly. Most of us are not yet aware about how diagnoses are read and interpreted.

Endometrial Hyperplasia involves alteration between two hormones. Estrogen is the hormone responsible for the development of female sex characteristics. It has a growth-stimulating factor that induces the thickening of the endometrial wall. It induces hyperplasia or cellular proliferation in order to prepare the endometrium for possible conception. The other hormone, progesterone, works as its regulator. Progesterone regulates the phase of cellular division in order to prevent excessive endometrial thickening. If conception does not occur, the endometrium sheds off in a form of menstruation.

The condition of Endometrial Hyperplasia occurs if the production of progesterone hormone becomes altered. If progesterone levels drop, the cellular division increases due to the absence of regulation. This excessively thickens the endometrial wall because of the continuous induction of estrogen. If this situation is prolonged, further complication may develop. Atypia or cellular abnormality arises if cells are exposed to too much levels of estrogen for longer periods. The diagnostic procedures for this disorder are vastly available in almost all hospital setting. The common problem is the understanding and interpretation of these diagnoses.

There are different grades to be understood in terms of endometrial hyperplasia. The common diagnoses that the health care providers give are as follows.

Relatively Benign. This diagnosis depicts Endometrial Hyperplasia but most likely belongs to simple to complex without atypia category. This is has the least risk among all the diagnosis. The chances for uterine cancer progression are very low.

Probable Benign. The hormone stimulation is present and cellular division is active. The hormone alteration is probably taking place. This condition is also less risky but medical follow up and continuous monitoring is necessary. This condition is usually alleviated by simple hormonal therapy.

Precancerous changes and cellular aggressiveness is observed. This type of diagnosis is very risky and requires immediate attention for further prevention. This stage is commonly experiencing a complex hyperplasia with evident atypia or abnormality. If this condition is not resolved, progression to uterine cancer might occur.

Cancerous. This diagnosis is the most dangerous of all. It indicates positive cancer cells proliferating in the uterine cavity. This requires immediate oncology management for treatment and prevention for its further progression. Such conditions usually lead to Atypical Adenomatous Hyperplasia, Endometrial Adenocarcinoma, Adenocanthoma, Endometrial intraepithelial neoplasia and invasive carcinoma.

These conditions are malignant and may require the patient to undergo hysterectomy or the removal of the whole uterus in order to prevent possible spread. This condition is very devastating for most of the females because their capacity for conception is greatly at risk. Knowing the interpretations of different diagnoses can serve as the life saving knowledge for us. This leads to early prevention which is primarily the most effective treatment for health anomalies especially cancer.

Article Source: http://www.drleetcmclinic.com/Health_Conditions/31.html

Endometrial Hyperplasia - The Types Involved

Endometrial Hyperplasia is one of the most feared diagnosis a patient can have. The public views this disorder as a stepping-stone towards uterine cancer. Misconception is running around in terms of this disorder. The disorder endometrial hyperplasia has different categories. The knowledge about these categories provides more background to the public especially for those who are anxious about this disease. Endometrial Hyperplasia is not entirely cancerous but can progress to uterine cancer.

Endometrial Hyperplasia is brought by the excessive thickening of the endometrium because of the hormonal alteration happening in the uterine cavity. The hormone responsible for this thickening is estrogen. It is a growth-stimulating hormone that induces the cellular hyperplasia in the area. The hormone responsible for the negation and regulation of this mechanism is Progesterone. It regulates the rate of cellular division in the area in order to prevent excessive thickening of the endometrium. If conception does not occur, the endometrial lining sheds and expelled from the body as menstruation. However, there are cases wherein progesterone decreases or becomes absent leading to the exponential cellular division in the endometrium. No regulation is present and estrogen still induces the cellular proliferation thus excessive thickening of the endometrial lining occurs.

Many people consider this ailment as a pre-cancer complication. It is however not entirely correct. There are four types of this disorders that have to be considered if we are going to link Endometrial Hyperplasia with cancer. Endometrial hyperplasia is divided into two categories, which are simple or complex hyperplasia without atypia (means cellular alteration or abnormality) and simple or complex hyperplasia with atypia.

Simple hyperplasia without atypia is the irregular and cystic proliferation of the glands in the uterine cavity. The cells become irregular in shape and pile up resulting to endometrial thickening. This is induced by the increasing estrogen and decreasing levels of progesterone. It is usually resolved by D and C and sometimes just by the normal menstruation. The structures of these cells particularly the organelles are also unchanged. This condition is considered the least dangerous type of endometrial hyperplasia. Complex hyperplasia is the crowding or budding of the glands. This condition is more irregular and usually resolved by pharmacotherapy and D and C but not with the regular menstruation. The induction of progesterone hormone forcibly shed the endometrium lining. These conditions are less likely to cause uterine cancer. Epidemiological studies indicate that less than 2% of the people diagnosed with these conditions develop uterine cancer.

Simple hyperplasia with atypia comprises several cellular alterations that are similar to cancer cells. The cells manifest extreme piling or stratification, altered organelles particularly the nucleus and continues to divide rapidly and exponentially. Complex hyperplasia with atypia also shows organelle alteration accompanied by cellular budding and extreme stratification. These conditions have the greatest risk for the development of uterine cancer. These conditions seldom respond to progesterone therapy and those who have had hysterectomies developed uterine cancer. Anxiety is common for those people diagnosed with this complication. The risks are well defined in this study and shows that not all people who contacts this hyperplasia develops uterine cancer.

Article Source: http://www.drleetcmclinic.com/Health_Conditions/31.html

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